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College of Veterinary Medicine

Philine Wangemann


PubMed search for Dr. Wangemann

Cochlea drawingWangemann, P. (2008)
Cochlear Homeostasis and Homeostatic Disorders.
In: J. Schacht, A.N. Popper and R.R. Fay (Eds) Auditory Trauma and Protection. Handbook of Auditory Research. Vol. 31, Springer, pp. 49-100.

Lang, F., Vallon, V., Knipper, M.,Wangemann, P. (2007) Invited Review: Functional significance of channels and transporters expressed in the inner ear and the kidney. Am. J. Physiol. Cell Physiol. 293: C1187-C1208.

Wangemann, P. (2006) Invited Review: Supporting sensory transductions: Cochlear fluid homeostasis and the generation of the endocochlear potential. J. Physiol. 576: 11-21.

Wangemann, P. (2002) K+ cycling and the endocochlear potential. Hearing Res. 165: 1-9.

Most cited publications:

(* > 30 citations; ** >100 citations)

* Wangemann, P., Itza, E.M., Albrecht, B., Wu, T., Jabba, S.V., Maganti, R.J., Lee, J.H., Everett, L.A., Wall, S.M., Royaux, I.E., Green, E.D., Marcus, D.C. (2004) Loss of KCNJ10 protein expression abolishes endocochlear potential and causes deafness in Pendred syndrome mouse model. BMC Medicine 2:30.

* Cohen-Salmon, M., Ott, T., Michel, V., Hardelin, J.P., Perfettini, I., Eybalin, M., Wu, T., Marcus, D.C., Wangemann, P., Willecke, K. and Petit, C. (2002) Targeted ablation of connexin26 in the inner ear epithelial gap junction network causes hearing impairment and cell death. Curr. Biol. 12, 1106-1111.

* Wangemann, P. (2002) K+ cycling and the endocochlear potential. Hearing Res. 165: 1-9.

* Marcus, DC, Wu, T. Wangemann, P., Kofugi P (2002) KCNJ10 (Kir4.1) potassium channel knockout abolishes the endocochlear potential. Am. J. Physiol. 282: C403-C407.

* Wangemann, P. and Schacht, J. (1996) Cochlear homeostasis. In: P. Dallos, A.N. Popper and R.R. Fay (Eds) The Cochlea. Handbook of Auditory Research. Vol. 8, Springer, pp. 130-185.

** Vetter, D.E., Mann, J.R., Wangemann, P., Liu, J., McLaughlin, K.J., Lesage, F., Marcus, D.C., Lazdunski, M., Heinemann, S.F. and Barhanin, J. (1996) Inner ear defects induced by null mutation of the isk gene. Neuron 17: 1251-1264.

** Wangemann, P., Liu, J. and Marcus, D.C. (1995) Ion transport mechanisms responsible for K+ secretion and the transepithelial voltage across marginal cells of stria vascularis in vitro. Hearing Res. 84: 19-29.

** Wangemann, P. (1995) Comparison of ion transport mechanisms between vestibular dark cells and strial marginal cells. Hearing Res. 90: 149-157.

* Marcus, D.C., Lui, J. and Wangemann, P. (1994) Transepithelial voltage and resistance of vestibular dark cell epithelium of gerbil ampulla. Hearing Res. 73: 101-108.

* Marcus, D.C., Takeuchi, S. and Wangemann, P. (1992). Ca2+-activated nonselective cation channel in apical membrane of vestibular dark cells. Am. J. Physiol. 262: C1423-C1429.

* Takeuchi, S., Marcus, D.C. and Wangemann, P. (1992). Maxi K+ channel in apical membrane of vestibular dark cells. Am. J. Physiol. 262: C1430-C1436.

* Takeuchi, S., Marcus, D.C. and Wangemann, P. (1992) Ca2+-activated non-selective cation, maxi K+ and Cl- channels in apical membrane of marginal cells of stria vascularis. Hearing Res. 61: 86-96.

* Wangemann, P. and Marcus, D.C. (1990). K+-induced swelling of vestibular dark cells is dependent upon Na+ and Cl- and inhibited by piretanide. Pflügers Arch. 416: 262-269.

* Wittner, M., Di Stefano, A., Wangemann, P., Nitschke, R., Greger, R., Bailly, C., Amiel, C., Roinel, N. and De Rouffignac, C. (1987). Differential effect of ADH on sodium, chloride, potassium, calcium, and magnesium transport in the cortical and medullary thick ascending limb of the mouse nephron. Pflügers Arch. 412: 516-523.

** Wangemann, P., Wittner, M., Di Stefano, A., Englert, H.C., Lang, H.J., Schlatter, E. and Greger, R. (1986). Cl- channel blockers in the thick ascending limb of Henle-structure-activity relationship. Pflügers Arch. (Suppl. 2) 407: S128-S141.